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In addition, as research intensifies, a more distinct link between diet and patients molecular alterations is emerging and becoming quantifiable, dispelling the previous conventional wisdom that linked phenotypic changes to dietary variation

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although at the advanced stages of the disease the dying neurones and compromised blood-brain barrier may act as instigators

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those with more advanced beta cell dysfunction may experience less benefit from GLP-1 therapy, as the medication relies on functional beta cells to stimulate insulin secretion