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Subsequently, CRM1 binds to nucleoporins, specifically Nucleoporin 214/Can protein, directing the complex toward the nuclear pore for transport, while fully spliced HIV-1 mRNAs can exit the nucleus without Rev [38]

These selective therapies can also be targeted to act specifically in immune cells within the TME [176], avoiding their antagonistic and undesired effects when affecting other cells, like tumor cells

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