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The strength of our model lies in the development of amyloid beta-like aggregates within human brain AD organoids and effectively demonstrates the preventive action mediated by ferrostatin-1

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HPLCMS analysis after solubilization with acidified methanol confirmed an almost stoichiometric conversion of 3,4- cis -leucocyanidin ( 6 ) into cyanidin ( 8 ), while the formation of the side products epidihydroquercetin ( 9 ), and quercetin ( 11 ) was reduced (Extended Data Fig