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In the heart and vasculature, ROS inflict oxidative damage on LDL particles, while inflammation accelerates atherosclerotic plaque formation

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At present, the key issue is not simply whether smoking changes OA risk, but how cigarette and EC exposure may influence the synovial-cartilage microenvironment and potentially contribute to more inflammatory or fast-progressing OA phenotypes

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I still seem to require an extra two hours of daily sleep, but the desperate despondency has largely evaporated