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Fluoxetine affects embryonic morphogenesis by inhibiting the activity of the Wnt signaling pathway and reducing cell proliferation, and activation of the Wnt signaling pathway partially mitigates the adverse effects of fluoxetine on embryonic morphology through a mechanism of action that is not directly related to the inhibition of serotonin reuptake in the synapse

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The combined effects of increased ROS production and inflammatory cytokine release can further impair neuronal function and viability, leading to the manifestation of depressive symptoms