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Both studies demonstrated that FSP1 acts as an NADH/NADPH-dependent CoQ 10 reductase that predominantly localizes to the plasma membrane via N-terminal myristoylationa posttranslational modification essential for its antiferroptotic activity

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10.1111/dom.15523 26 GuerdjikovaA

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This physiological dysregulation could signal an interplay in patients who have kidney disease and other chronic conditions like cardiovascular disease, diabetes and obesity, which are highly prevalent in this population