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Importantly, biological markers revealed that e-cigarette users exhibited elevated levels of pro-inflammatory molecules, including cytokines such as interleukin-1 and tumor necrosis factor-, compared to non-smokers
We found that adverse effects of cigarette smoke on surfactant were induced by tar, which is the product of burning, and other major components of cigarette smoke (nicotine, acetaldehyde, and isoprene) did not cause surfactant disruption
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