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5d) therefore suggests a less efficient interaction between E170X mutants and HEDS-derived GSSEtOH in accordance with the scaffold model and the hypothesis that GSSEtOH formation is catalysed by ScGrx7

contortus although has identified multiple candidates, however, their use as potential therapeutics can be hindered due to lack of knowledge on their mechanisms of action [5155]

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doi: 10.3390/gels8110747, 280