Similar content being viewed by others Introduction Type 2 diabetes mellitus (T2DM) is a prevalent metabolic disorder worldwide characterized by high blood glucose levels, with insulin resistance and pancreatic cell pathology as the main symptoms, accompanied by pathophysiological changes, such as oxidative stress and chronic inflammation 1,2,3
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Melatonin protects against QA-induced damage to the pyramidal cell layer through direct free radical scavenging rather than receptor-mediated signalling, suggesting that KP hyperactivity may simultaneously increase oxidative burden and diminish endogenous neuroprotection [168]
A further important safety concern is the unknown interaction profile of these peptides with prescribed medications, including insulin, antihypertensives, and hormonal therapies
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