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Although basal XOR expression is typically low in humans, various factors such as hypoxia, ischemia-reperfusion injury, interleukin-1 (IL-1), interleukin-6 (IL-6), tumor necrosis factor-alpha (TNF-), and corticosteroid treatment can markedly enhance XOR transcription

This might regulate cellular viability and could not involve specific plasma membrane receptors

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Individual Adjustment : Customizing protocols based on individual responses rather than using uniform dosing across all participants

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