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This suggests that the body is conserving free L-carnitine to be able to bind to long-chain fatty acids, but it also shows an elevated excretion of short-chain acetyl-CoA

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To systematically assess whether metabolic and mitochondrial dysfunction is associated with the pathophysiology of post-exertional malaise 23,24,25,26 , we measured mitochondrial respiration and metabolomic signatures in skeletal muscle before and one day after the induction of post-exertional malaise