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Reactive astrocytes exacerbate inflammation by releasing additional cytokines, contributing to a feedback loop that worsens neuronal injury ( The interaction between excitotoxicity and neuroinflammation is critical for the development of long-term neurodegeneration

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in DEM treated cells the growth is delayed until the higher level of nuclear GSH is reached, which occurs at 4872 h, while in the BSO treated cells the maintained high level of nuclear GSH between 48 h72 h results in a prolonged cell growth when compared to control

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