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[DOI] [PMC free article] [PubMed] [Google Scholar] [Retracted] 22.Mahmoodi M, et al

However, under pathophysiological conditions, especially during APAP overdose, excessive NAPQI accumulates and binds to cellular proteins, also triggering oxidative stress, leading to hepatocellular damage and the release of damage-associated molecular patterns (DAMPs), contributing to further liver injury and inflammation

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The reduction in SLC7A5 protein levels, without a corresponding decrease in mRNA levels upon YTHDF1 knockdown, indicates that YTHDF1 could be involved in regulating the stability or translation of SLC7A5 protein

Group I mGluRs include mGluR1 and mGluR5, which are associated with the activation of phospholipase C [11]